May be aOnly reason Back Molecular confusion It stimulates itUlcerrerers: The RNA particles and proteins in nerve cellsA process that usually occur in response to stress conditions, but sometimes becomes chronic, which is a Traffic block between nucleus and cytoplasm That Slavery Manda i Neurons. It seems that this new combination theory can be explained for the first time, all the most different molecular changes of the disease, the researchers of the Arizona State University, with an article Released In Alzheimer’s & Dementia Magazine.
Their work, a decision The systematic study of studies in the literature And the biomass analysis of existing databases, which provides a unique frame, in which various aspects of the disease are amyroid plates, the complications of the AAU protein, inflammation and various cell dysfunctions to design a disconnected fragment of Alzheimer.
Researchers led by Paul Coleman say all this Changes May be due to a reason: so -sized Particles of depressionAggregates of proteins that are temporarily formed by RNA Response Alo Depressive cellular For example, genetic changes, inflammation, exposure to pesticides, viruses and atmospheric pollution were unleashed. These particles suspend the essential processes when the cell is recovered, and dissolves when the stress is attracted.
Infected However, the provider of Alzheimer The particles are continuing E They are chronicEnding Sprinkle with key molecules E. Disruption to their movements Cell fetus and from. This interruption of traffic Prevents the production of essential proteinsMolecular switches that control the operation of a thousand genes ie, Incitement Layered Clinical events Alzheimer. These Changes They happen a lot In the beginningToo Prima From Appearance Regular Symptoms From Sickness Like the problem of amyide plates or A AU. Identify and confront the formation of pathological pressure particles in the early stages This may significantly arrest or delay the onset of symptoms.
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